Longer telomeres may shield mice from age-related human diseases

(JCI Journals) Researchers in Deepak Srivastava's laboratory at the Gladstone Institute of Cardiovascular Disease hypothesized that mice may be protected from age-associated human diseases due to the relatively longer length of their telomeres, the regions at the end of chromosomes that help guard against deterioration. In work published this week in the JCI, the researchers used mice with shortened telomeres to examine a genetic defect that causes an age-associated congenital heart disease in humans.

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